Immune cell-specific transcriptional profiling highlights distinct molecular pathways controlled by Tob1 upon experimental autoimmune encephalomyelitis.

2a–d .) However, a negative trend was detected for IgG levels in Tob1-knockout B cells with borderline significance (two-tailed T-test, P = 0.068) ( Fig. 2c ).

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Thus, we extended our analysis to nominally significant genes but very little overlap was again highlighted at the 3 time points investigated, confirming that most of the genes regulated by Tob1 are cell-specific ( Fig. 5a ).

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