Retention of normal glia function by an isoform-selective protein kinase inhibitor drug candidate that modulates cytokine production and cognitive outcomes.

However, these data must be interpreted with caution because the in vivo results with Aβ plaque volume did not reach statistical significance, and in vitro results in the BV2 cell line with bioparticle phagocytosis cannot be directly equated to Aβ phagocytosis in vivo.

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KI mice treated with MW150 showed a trend of reduced GFAP staining in the cortex compared to KI + veh-treated mice, but this difference did not reach significance (Fig. 2b ).

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