MitoQ supplementation prevent long-term impact of maternal smoking on renal development, oxidative stress and mitochondrial density in male mice offspring.

Maternal MitoQ administration ameliorated MCP-1 expression although this did not reach statistical significance (Fig. 3a ); whereas CD68 expression and F4/80 protein level were normalised in the SEMQ offspring (P < 0.01 vs SE, Fig. 3d ; P < 0.05 vs SE, Fig. 3e ).

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Maternal MitoQ supplementation showed a trend to normalization of urinary albumin-to-creatinine ratio.

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