Prolonged restraint stressor exposure in outbred CD-1 mice impacts microbiota, colonic inflammation, and short chain fatty acids.

Aerococcus demonstrated a significant positive association with iNOS, IL-1β, and histopathology scores (p<0.001), but a negative trend with propionic acid (p<0.05).

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In the absence of infection, the mean Lactobacillus relative abundance in stressor-exposed animals was lower than in control mice, but this difference did not reach statistical significance.

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