Langerin+ DCs regulate innate IL-17 production in the oral mucosa during Candida albicans-mediated infection.

Of the three identfitied IL-17A-producing cellular subsets, the TCRβ + subset was most strongly affected, but also the TCRγδ + and ILC populations showed a clear trend towards reduced IL-17A production in IL23-deficient mice compared to WT controls.

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This had only a limited impact on IL-17A production by the three CD90 + celluar subsets that did not reach statistical significance ( S9B Fig ).

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