Pomalidomide Ameliorates H₂O₂-Induced Oxidative Stress Injury and Cell Death in Rat Primary Cortical Neuronal Cultures by Inducing Anti-Oxidative and Anti-Apoptosis Effects.

POM 500 nM demonstrated a trend to lower H 2 O 2 generation that failed to reach statistical significance ( p = 0.15).

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After 0.5 h of H 2 O 2 insult, intracellular ATP levels were lower across all injured groups with and without POM 50 and 500 nM; however, these differences did not reach statistical significance ( Figure 2 b).

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