Metabolic stress is a primary pathogenic event in transgenic <i>Caenorhabditis elegans</i> expressing pan-neuronal human amyloid beta.

Even though we observed a trend towards lower levels of insoluble protein in the Metformin-treated GRU102, the difference did not reach statistical significance ( Figure 6C ), possibility due to the variability of the gel.

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We further found that Metformin-treated GRU102 showed a trend towards lower levels of protein aggregates.

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