Ginsenoside Rg3 micelles mitigate doxorubicin-induced cardiotoxicity and enhance its anticancer efficacy.

In response to P-Rg3 treatment, these indices tend to increase, but this did not reach statistical significance compared with the levels in the DOX group ( Figure 3(A,D–F) ).

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The amounts of proteins involved in oxidative phosphorylation, as assessed by immunoblotting, showed a trend similar to that in vivo ( Figure 6(G–J) ).

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