Deficient DNA base-excision repair in the forebrain leads to a sex-specific anxiety-like phenotype in mice.

In support of this notion, male KO mice also showed a trend towards an increased latency to enter the bright compartment of the testing apparatus, albeit this trend did not reach statistical significance ( t (9) = 1.55, p = 0.15) (Fig. 3 A).

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The corresponding ventral structures showed a slight reduction in XRCC1 levels that failed to reach significance (Fig.

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