A1, but not A2, was increased in ROP samples as compared to controls yet this did not reach statistical significance (Fig.
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Targeting proliferative retinopathy: Arginase 1 limits vitreoretinal neovascularization and promotes angiogenic repair.
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Mice lacking A1 in endothelial cells exhibited a significant reduction in RNV tuft formation at P17, while the AVA showed a trend towards improved repair that was not statistically significant (Fig. 7A–C ).