Ceruloplasmin-Deficient Mice Show Dysregulation of Lipid Metabolism in Liver and Adipose Tissue Reduced by a Protein Replacement.

Despite a clear trend of protein’s level reconstitution in CpKO mice, the large variability of rescued levels made not significant the statistical evaluation of the two-ways ANOVA for the interaction of genotype/treatment ( Figure 2 c,d).

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Interestingly, the treatment with Cp showed a trend toward a limitation in body weight increases in the CpKO mice ( Figure 1 b). 2.2.

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