Pitavastatin treatment remodels the HDL subclass lipidome and proteome in hypertriglyceridemia.

Statin treatment (D180) exerted a marked trend to increase in lipid abundance in (total) HDL (normalized to apoAI), except for DAG and TAG, which trended lower (NS) ( Table 2 ).

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With minor exception, molar ratios of TAG/PC and DAG/PC were diminished poststatin, with a strong trend to normalization of core CE/TAG ratios.

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Poststatin lipidomic profiles for TAG, DAG, LPC, LPC(O), PE, and PI/PC showed a trend to decrease across HDL subclasses, whereas those of SM, PC(P), PC(O), PE(P) PE(O), and GM3/PC trended to increase ( Fig. 1 ).

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