showed a trendP = 0.022
This risk haplotype was also statistically significant in SZ patients ( P = 0.0011) and showed a trend of association in BD patients ( P = 0.022).
This risk haplotype was also statistically significant in SZ patients ( P = 0.0011) and showed a trend of association in BD patients ( P = 0.022).
Model 2 showed a trend toward improving upon model 1 after correction for multiple tests, R change 2 = .18 , p = .022.
However, Locator-type attachments showed a trend towards lower levels of satisfaction, which in comparison with bars ( p = 0.022) was statistically significant prior to the Bonferroni correction for multiple testing ( Table 2 ).
The difference between BWUref and BWUref + 15% showed a trend towards a lower activity when unloading with an additional 15% (p = 0.022).
In addition in the MD Anderson cohort patients in Luminal A ( P = 0.085) and Luminal B ( P = 0.068) subgroups showed a trend towards significance in terms of improved CSS, while in our cohort this applied only for Luminal A vs. non-Lumina A ( P = 0.022).
The patient bearing the combining signatures showed a trend toward shorter OS ( P = 0.022, Fig. 2C ) and EFS ( P = 0.07, Fig. 2D ) than other patients.
Discussion Our analysis of 6155 patients (mean follow-up: 5.47 years) found that raloxifene showed a trend toward lower TKR risk vs. bisphosphonates in crude analysis (HR 0.62, 95% CI 0.42–0.93, p = 0.022), but this association became non-significant in the traditional adjusted Cox model (aHR 0.71, 95% CI 0.47–1.07, p = 0.102).
In the LIN4 - CD45 dim CD34 + , viral load showed a trend of a negative correlation with the expression levels of CD49f (R= -0.420, p = 0.022), and the frequency of CD4 and CD8 HLADR + CD38 + was negatively associated with the expression of CD31 (R= -0.465, p =0.014 and R=- 0.584, p = 0.001 respectively).
We classified patients according to the UPCR, and with the increase of proteinuria, the serum creatinine of the patients showed a trend of gradual increase and the difference between the different groups was statistically significant (P=0.022).
In PVF, we observed a statistical difference in the number of switches, and in SVF, MCI-NC showed a trend toward producing more switches ( p = 0.022 and p = 0.065, respectively; Table 3 ).
ACWR TD W3 showed a trend toward an association with injury risk (χ 2 (1) = 5.24, p = 0.022; R 2 McF = 0.094; AUC = 0.675; OR = 236.33, 95% CI [0.91, 61,385.02], p = 0.054), while ACWR TD W4 also demonstrated a marginal trend ( p = 0.076; AUC = 0.665).
Results Results showed a trend towards susceptibility to cutaneous lesions for alleles HLA-DRB1*13 ( P =0.0228; Pc =0.3420; OR=1.66; 95%CI=1.08 – 2.56), HLA-B*35 ( P =0.0218; Pc =0.6758; OR=1.67; 95%CI=1.08 – 2.29) and HLA-B*44 ( P =0.0290; Pc =0.8990; OR=1.67; 95%CI=1.05 – 2.64).
HDAC5 mRNA expression in classic monocytes of MD patients showed a trend towards an increase compared to HC ( p = 0.023; FDR correction q = 0.092) (Fig. 5 A).
Lower SES (more deprived) patients showed a trend toward increased AT in the multivariate Cox model, with a hazard ratio (HR) of 1.000 (95% CI: 0.999–1.000, p = 0.023).
Specifically, they gave less negative evaluations of the negative scenes (this was significant for the category Distress t (11.36) = 3.64, p < .001, with trends in the same direction for scratch ( p = .034) and yawn ( p = .014)) and less positive ratings following positive scenes (which was significant for grooming t (11.36) = 3.44, p = .001 and showed a trend in play ( p = .023).
Lower SES (more deprived) patients showed a trend toward increased AT in the multivariate Cox model, with a HR of 1.000 (95% CI [0.999–1.000], p=0.023).
During a mean follow-up period of 34.6 months, the high-threshold group showed a trend toward a higher incidence of HB capture threshold of ≥2.5 V (50% vs. 14%; P=0.023), HBP abandonment (29% vs. 8.6%; P=0.091), lead revision (21% vs. 2.9%; P=0.065), and clinical events (all-cause death, heart failure hospitalization, and new-onset or progression of atrial fibrillation; 50% vs. 23%; P=0.089) than the low-threshold group.
FMD showed a trend toward improvement in the IHC group, being significant when normalized for baseline artery diameter ( p = 0.023; η p 2 = 0.150) between Pre and Post 2.
Results In vivo, the traditional IVIM model showed a trend toward higher f with increasing TE in the liver ( R = 0.46, p = 0.023), but not in the kidney cortex ( R = −0.067, p = 0.76) or medulla ( R = 0.039, p = 0.86).
A region in the vmPFC showed a trend of enhanced BOLD activity 1 month after compared with before CAT ( Supplementary Fig. 5 a ; cluster size = 58 voxels, max Z value = 3.21, cluster corrected P = 0.023; not significant following Bonferroni correction), similarly to the short-term change.
Interestingly, prior trastuzumab exposure did not significantly affect median PFS [12.57 versus 8.05 months, p = 0.541, Figure 5(a) ], yet a longer duration (⩾6.37 months) showed a trend toward improved PFS [12.02 versus 5.32 months, p = 0.023, Figure 5(b) ].
In Ptprj KO cells, the ERK1/2 phosphorylation sites T202 and Y204 were significantly increased after 5 min, showed a trend toward elevated phosphorylation after 10, 20, and 30 min (Figure 1J,K ) and an increase in phosphorylation ( p = .0234) when quantifying the entire period (Figure 1L ).
Then, HOM generated with differentiated MSCs showed a trend to show a higher number of CD34‐positive cells than positive control, which reached statistical significance in the case of HOM‐dADSC ( p = 0.0238). 4.
Predictive factors for extreme responders When subgroups were created to identify extremely poor (≤ 7 cycles, F7) and good (≥ 20, M20) responders, the M20 group showed a trend similar to that of the M10 group with higher numbers of M1a stage patients (42.3% vs. 20.8%; P = 0.024), a lower incidence of brain metastasis (23.1% vs. 42.6%; P = 0.069), and lower levels of TS expression (median H‐score 5.0% vs. 50.0%; P = 0.030) (Fig 2 b, Table 2 ).
On the other hand, denervated Sol muscles from the KO mice showed a trend toward a reduction in absolute force that became significant when force was normalized for muscle mass ( P = 0.024 vs.