Excerpts

Of note, the association between HSS-KRES and change in WOMAC function at 6 months was trending towards significance in the multi-variable model [ß: − 0.03, 95% CI − 0.07 to 0.00, p = 0.069].

Indeed, there was a marginally significant difference in agency index between the synchronous and asynchronous conditions (χ 2 = 3.32, df = 1, n = 20, P = 0.069), if another statistical criterion was applied to the data.

However, it is noteworthy that the presence of detectable ctDNA post-treatment showed a trend towards worse OS (HR 7.13, 95% CI 0.86–59.3, P = 0.069).

However, the VATS group had a better RFS rate than the thoracotomy group, albeit with marginal significance ( p = 0.069) (Fig. 3 B).

In contrast, SSRI use (SSRI use, n = 10 vs no SSRI use, n = 7) was marginally significant (F (1,14) = 3.890, p = 0.069) such that participants with PTSD on SSRIs had 27% lower [ 18 F]FEPPA V T compared to participants not on SSRIs and showed no difference from HC (p = 0.837).

Though the latter was only borderline significant on log-rank testing for PFS ( p = 0.069), this was likely due to the small sample size of the MSKCC validation cohort (N = 83) and the presence of confounding variables (PD-L1, TMB).

The soluble Aβ42 levels in both AAD-66 and SI treated groups compared with the vehicle treated group showed a decreasing trend (p = 0.0692, p = 0.2451, respectively) (Fig. 3E ).

Although adult Scn2a +/− mice showed a trend towards significant slower acquisition latencies on day1–4 (two-way repeated measures ANOVA, F(1, 29) = 3.56, p = 0.0693 for genotype (borderline significance); F(5, 145) = 90.04, p < 0.0001 for day; F(5, 145) = 1.1, p = 0.3813 for genotype × day interaction), their performances were comparable to wild-type mice during the probe tests on days 5 and 12.

Stress did not significantly increase the number of spines in LAT neurons from males, though there was a strong trend (stress × distance interaction, p = 0.0696, F(29,754) = 1.425, two-way RM-ANOVA, N = 14 control, N = 14 stress).

A test for interaction across subgroups according to number of children (0, 1, 2, 3 or more children) showed a decreasing trend in the odds ratios (p = 0.07).

SPR for Brother John were also significantly correlated with all other rates (from SMT online pre-lab: r (45) = 0.37, p = 0.014) to clapping: r (53) = 0.57, p < 0.001), except walking ( r (49) = 0.26, p = 0.076), and those for Twinkle only fell short of significance with walking rate ( r (50) = 0.26, p = 0.070).

Extinction A 2 × 3 × 2 rmANOVA using Context (CTX+, CTX−), Trial (start, middle, end) as within-group variables and Group (novel exploration, control) as a between-group variable showed an effect of Trial, F(1.47,77.63) = 9.45, p < 0.001 G-G corrected, η 2 = 0.15 and a near-significant effect of Context, F(1,53) = 3.51, p = 0.07.

Treatment of polarized ARPE-19 cells with 100 nM ZK for 3 h showed a trend (p = 0.07) to increase BECN1 expression in low autophagy-/UPR-related gene-expressing cells (Fig. 6 c) but significantly decreased BECN1, MAP1LC3B, mTOR, ATF6, DDIT3, EIF2AK3 and HSPA5 expression (Fig. 6 a,c,d,f,j,k,m) in high autophagy-/UPR-related gene-expressing cells.

For CG, as regards the frontal ROI, a post-hoc analysis (Tukey HSD, MSE = 52.49, df = 43.28) showed a trend towards increased activation for intentional harm compared with unintentional harm ( p = 0.07).

However, the higher relative abundance of Prevotella bivia in women with SPE was only marginally significant after adjustment for BMI ( P = 0.07) in the confirmation cohort, suggesting that the role of Prevotella bivia in SPE may be modified by BMI.

Spring weight of male individuals varied significantly between crosses ( F 3, 334 = 18.37, p < 0.001) and habitats ( F 1, 334 = 72.14, p < 0.001) with a marginally significant interaction effect ( F 3, 334 = 2.40, p = 0.07).

The unadjusted result was of borderline significance on univariate analysis (p = 0.07) (Table 2 , Fig. 3 ).

However, when very preterm adolescents with moderate-severe BPD were compared to very preterm ones without BPD, the first group showed a trend towards worse left ventricle function: less shortening fraction (37.5 ± 3.2% vs. 40.7 ± 4.9%, p = 0.07), less ejection fraction (68 ± 4.4% vs. 71.2 ± 5.4%, p = 0.06) and a greater E/E’ ratio (6.4 ± 0.8 vs. 4.9 ± 1.04, p = 0.01).

With respect to epidemiological characteristics with binomial values, Fusobacterium was associated with alcohol consumption and KRAS mutation ( P = 0.088 and P = 0.094), and Parvimonas was associated with tumor location and KRAS mutation, although the difference was only marginally significant ( P = 0.070, Table 4 ).

suis -like clinical signs was higher (17.7%), and compared with the prevalence in the Amox group, the difference was close to being statistically significant ( P = 0.07).

Accordingly, considerable differences were found in the overall amount of VOCs released (AUC; except for (Z)-3-hexenyl acetate that showed a trend: p = 0.07) and in the maximum intensity released (I max , except for ethyl maltol).

1a ) and the midday leaf water potential showed a decreasing trend ( R 2 = 0.60, p = 0.07) with declining tree health (Fig. 1b ).

7 a, right), in the left hemisphere, similar to the whole brain, the Pseudo-Lesion Group exhibited a marginally significant negative correlation with modularity (r = − 0.23, p = 0.07), whereas the Real-Lesion Group did not (r = − 0.12, p = 0.28).

A positive trend between R 0 and FEV 1 was observed (p = 0.07).

In the FST (F2.45 = 3.242, p = 0.048, n = 16), CUS rats showed a trend toward more time spent immobile over the 6-min test period than rats of the non-CUS control group (p = 0.070).