Barely Significant
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“marginally significant”

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Sighted at

p>=0.1

Listed by Hankins (2013) · Otte et al. (2022)

In the literature

marginally significantP < 0.051.0× alphagold
lanceolatum MRF neighborhood and human chromosomes 11 and 12 (binomial test, 11: P < 2 × 10 −3 , 12: P < 1 × 10 −2 ; Barnard’s exact test, 11: P < 1 × 10 −10 , 12: P < 2 × 10 −8 ; supplementary information 6 c – d , Supplementary Material online), whereas there was no significant association observed with chromosome 1, and only a marginally significant association with 19 in one of the tests (Barnard’s exact test, P < 0.05).
marginally significantp = 0.051.0× alphagold
The pattern of results was highly similar: a significant main effect of BART condition: F (2,343) = 8.00, p < 0.001, η p 2 = .05, no main effect of Raven’s SPM: F (1,343) = 0.16, p = 0.69 η p 2 < .001, a significant BART condition by Raven’s SPM interaction: F (2,343) = 1.56, p = 0.02, η p 2 = .02, and a marginally significant three-way interaction between BART condition, Raven’s SPM and sex: F (2,343) = 3.02, p = 0.05, η p 2 = .02.
marginally significantP = .051.0× alphagold
There was a statistically significant moderate positive correlation between total brain volume (%ICV) and EZ-RPE volume in PD group (Pearson correlation coefficient 0.482, P = .04), normal cognition control group (Pearson correlation coefficient 0.514, P < .01) and a marginally significant moderate positive correlation in AD dementia group (Pearson correlation coefficient 0.526, P = .05) ( Fig 3 ).
marginally significantp = .051.0× alphagold
While we did not observe significant interactions with genotype group within the latter latency window (Congruency×Genotype: p>.05, Congruency×Genotype×Site: p>.05), we did observe a (marginally significant) Congruency×Genotype interaction in the earlier (600 and 800 ms) time window (F(1,47) = 4.03, MSe = 46.38, p = .05; Congruency×Genotype×Site: p>.05).
marginally significantP = 0.051.0× alphagold
The combination of the two samples, authorized by the absence of any significant clinical difference between them, showed a marginally significant association of the C allele (AFBAC, RA index cases 64.8% versus controls 58.2%, P = 0.05; TDT, 56.1% of transmission, P = 0.09) and a significant excess of the C-allele-containing genotype in RA index cases compared with controls (173 RA index cases versus 157 controls, P = 0.02).