Barely Significant

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borderline significantp = .051.0× alphagold
Results showed no significant relations between mothers’ prenatal socioeconomic or social support adversity and offspring’s parasympathetic nervous system trajectories, but there was a statistically significant relationship between social support adversity and offspring’s heart rate trajectories (p<.05) and a borderline significant relationship between socioeconomic adversity and offspring’s sympathetic nervous system trajectories (p = .05).
marginally significantp< 0.051.0× alphagold
With the exception of the non-significant difference between the DMN and the single system ( X 2 = 2.54 p = 0.1) and the DMN and the δ-Single between-network connectivity (X 2 = 1.53 p = 0.2) which was likely explained by the relatively large overlap between the DMN and the single system, the results showed that the difference between the fc-behavior correlation coefficients of the two sets of networks was significant (or marginally significant) (Single-SMN: X 2 = 3.6, p< 0.05; Single-VN: X 2 = 5.69, p< 0.05; β-Single –DMN: X 2 = 4.78 p< 0.05; β-Single –SMN: X 2 = 6.64 p< 0.01; β-Single –VN: X 2 = 9.56 p< 0.01; δ-Single – SMN:: X 2 = 3.38 p = 0.06; δ-Single – SMN:: X 2 = 5.52 p< 0.05).
borderline significantp = 0.051.0× alphagold
Sex-specific linear regression analyses, while not reaching significance level of 0.05, indicated that the associations between As exposures and Alu, LINE-1 and LUMA were positive among male newborns (N = 58) but negative among female newborns (N = 43); tests for sex differences were borderline significant for the association of cbAs and mbAs with Alu (p = 0.05 and 0.09, respectively) and for the association between maternal uAs and LINE-1 (p = 0.07).
showed a trendp <0.051.0× alpha
3C ) showed a trend toward increased tau 4R expression (exon inclusion) in 2-month-old ADNP+/− mice compared to ADNP+/+ controls ( p <0.05, one tail, t-test comparing only tau 4R expression), which disappeared in older, 9-month-old mice, showing similar tau species expression in the ADNP+/+ and ADNP+/− mice ( Fig. 3C ).